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Therefore, there is an increase in oxidative stress, deregulation of calcium homeostasis, inadequate activation of the renin-angiotensin-aldosterone system, mitochondrial dysfunction, lipid peroxidation, post-translational modification of proteins and the activation of several inflammatory and signaling pathways involved in the promotion of cardiac oxidative stress that mediates cellular and extracellular lesions

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Adewole OO, Omotoso BA, Ogunsina M, Aminu A, Odeyemi AO, Awopeju OF, Ayoola O, Adedeji T, Sogaolu OM, Adewole TO, et al