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The potential causes may be low blood pressure, gastric issues, and cardiac arrest (not confirmed yet)

Cardiac-specific deletion of mitochondrial complex I subunit NDUFS4 protein resulted in accumulation of NADH in mitochondria and elevation in NADH/NAD + in rodent heart, which correlated with the accelerated development of heart failure induced by pathological pressure overload or isoproterenol (42, 47)

Angiogenic factors have emerged as the most specific biomarkers of PE ever described and have recently been incorporated as essential components in the prediction, diagnosis and prognosis of PE (88)