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10.1515/hsz-2017-0131 10.1515/hsz-2017-0131 [DOI] [PubMed] [Google Scholar] Glorieux C, Calderon PB (2018) Catalase down-regulation in cancer cells exposed to arsenic trioxide is involved in their increased sensitivity to a pro-oxidant treatment

Via this metabolic adaptation, cells can continually generate TCA metabolites, such as -KG and citrate, which are converted to cytosolic acetyl-CoA for lipid biosynthesis (Fig
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Konosic et al., 2019), and vasomotor tone occurred through BPC 157-specific activation of the Src-caveolin-1-eNOS pathway (Hsieh et al., 2020)

In general, persistent systemic inflammation in the lungs and localized neuroinflammation in the brain, are significant triggers of depression and cognitive deficits[164]
