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Takeda and Akira, 2015)
272 In general, the pathological process of hepatic I/R injury involves damage to sinusoidal ECs, liver cells, hepatic stellate, and other cells
In the FTIR-spectra of GSSG in presence of complex 1 (Fig
GSH behaves as redox sensor at the DNA synthesis onset by maintaining nuclear architecture providing the appropriate redox environment for DNA replication and safeguarding DNA integrity (72) and is a key regulator of epigenetic events critical in cell proliferation regulation and cellular resistance to apoptosis (73)
