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These findings all indicate that ferroptosis promotes the development of DCM through abnormal iron metabolism, lipid peroxidation, ROS production, and enhanced cardiomyocyte inflammation (Figure 1)
However, it must be emphasized that the hypoxic responses, metabolic reprogramming, and accumulation of mutations potentially induced by this network draw upon concepts from certain areas of cancer research
Kaya, C., Akram, N
Lin et al., 2015)
