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has demonstrated that it reduces myocardial hypertrophy and fibrosis in ventricular remodeling (93)

and our group reported that macrophage and VMSCs apoptosis is necessary to reduce macrophage burden and stabilize the atherosclerotic plaque within the developing lesion in Apo E / mice [67, 68]
Borrelli 2000

In Parkinsons disease, abnormal magnesium levels are linked to transporter dysfunctions, suggesting that supplementation could stabilize these transport mechanisms and potentially slow disease progression (48)