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Upon pro-apoptotic stress, activated BAX and BAK induce mitochondrial outer membrane permeabilization (MOMP)

Here, TNF- leads to the accumulation of collagen and can lead to the remodeling of the matrix, resulting in fibrosis through the induction of apoptosis [23,24]

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Concurrently, the mitochondrial signature lipid cardiolipin facilitates Drp1 oligomerization, synergistically enhancing coordinated constrictive remodeling of both OMM and IMM ( Recent breakthroughs have been made in the molecular mechanisms surrounding mitochondrial dynamics