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[DOI] [PMC free article] [PubMed] [Google Scholar] 67.Mikus D, Sikiric P, Seiwerth S, Petricevic A, Aralica G, Druzijancic N, et al

In these models, fibroblasts efficiently migrate, proliferate, and deposit matrix components to facilitate initial wound closure, yet they do not persistently differentiate into highly contractile, matrix-overproducing myofibroblasts

Co-treatment protocols demonstrate enhanced VEGFR2 signalling when TB-500 is present, suggesting cytoskeletal reorganisation facilitates receptor clustering and downstream cascade amplification

J Neurol Neurosurg Psychiatry 31: 1922, 1968