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Researchers have proposed that AOD9604 may stimulate lipolysis directly within adipose cells and indirectly increase overall energy expenditure, potentially through modulation of 3-adrenergic receptor (3-AR) expression a receptor type understood to be central to fat breakdown in adipocytes

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However, the narrow therapeutic window of this intervention necessitates a better understanding of the intricacies of APAP-induced liver injury for the development of additional therapeutic approaches that can benefit late-presenting patients

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