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Pre-clinical animal models also shed new light on possible mechanisms of CIPN, including abnormal cytoskeletal architecture, disrupted axonal transport, mitochondrial dysfunctions, altered neuronal excitability in peripheral neurons, and neuroinflammation
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10.1016/j.redox.2016.12.035 10.1016/j.redox.2016.12.035 [DOI] [PMC free article] [PubMed] [Google Scholar] Sies H (2023) Oxidative eustress: the physiological role of oxidants
However, juvenile or adult onset manifestations characterized mainly by ataxia, dementia, and psychosis have been described [2, 4]
