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At a cellular and molecular level, more work is needed to explore the mechanisms of how L-carnitine could exert its action, whether due to its impact on mitochondrial energy metabolism, its potentially anti-ischemic qualities, or its effect on left ventricular remodeling

There are a bunch of other inborn errors of metabolism, including short-chain acyl-CoA dehydrogenase deficiency and medium-chain acyl-CoA dehydrogenase deficiency.[ref] Lets look at the inborn errors of metabolism involving carnitine: CPT2 gene: The CPT2 gene encodes the enzyme that moves fatty acids attached to carnitine into the inner membrane of the mitochondria