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This disruption is characterised by increased permeability evidenced by tracer extravasation, blood product accumulation, and focal tight junction widening and is accompanied by sustained inflammatory signalling, likely driven by ongoing extracellular matrix remodelling and perivascular dysfunction

MacFabe DF, Cain NE, Boon F, Ossenkopp KP, Cain DP

Liu J, Head E, Gharib AM, Yuan W, Ingersoll RT, Hagen TM, Cotman CW, Ames BN: Memory loss in old rats is associated with brain mitochondrial decay and RNA/DNA oxidation: partial reversal by feeding acetyl-L-carnitine and/or R-alpha -lipoic acid

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