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Although speculative, it is tempting to consider that the detrimental interaction of A 142 with mitochondria may be related to its evolutionary origin as an enslaved proteobacteria and therefore may experience some off-target effects of AMPs (Roger et al., 2017

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[29] However, a unified theory of how catenin drives target gene expression is still missing, and tissue-specific players might assist catenin to define its target genes