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The inflammatory adipokines resistin and visfatin compromise metabolic health through multiple mechanisms: disrupting IR activation, amplifying inflammatory protein production, and perturbing glucose homeostasis (360)

Future investigations should incorporate high-resolution joint-tissue single-cell transcriptomics, long-term OA models, and clinically stratified cohorts to further elucidate the diagnostic utility and mechanistic function of LPCAT3 in OA pathogenesis

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