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This model needs to be further tested
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Specifically, in ischemic/high-ROS environments, BACH1 tends to be degraded and acts to suppress angiogenesis by inhibiting the Wnt/-catenin signaling pathway and recruiting HDAC1, among other mechanisms
(For more information on adduct formation and other detrimental effects of acetaldehyde, see the article by Quertemont and Didone, p
