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Moreover, siRNA knockdown of p97/VCP in HEK293 cells significantly increased the amount of ATP7A T994I retained in the TGN, directly implicating the aberrant interaction between these two proteins in the mislocalization of ATP7A T994I at the plasma membrane (Yi et al., 2012)

From antioxidant depletion to mitochondrial dysfunction, oxidative stress orchestrates a cascade of cellular disruptions [93]

ACEIs and ARBs are central to the treatment of hypertension, heart failure with reduced ejection fraction, and chronic kidney disease

Association between mitochondrial dysfunction and severity and outcome of septic shock