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In contrast, it would appear that the neurologic effects of cobalamin deficiency are due to metabolic disturbances unrelated to purine synthesis because the neurologic deficits may develop independently of the hematologic abnormalities, and folate supplementation will not prevent or reverse these deficits

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Unlike full-length growth hormone, it appears to function without significantly altering insulin levels or IGF-1 pathways, making it a more specialized tool for metabolic studies

Due to its experimental status, individuals should approach 5-AMino-1MQ with caution