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The hyper-inflammatory response caused by SARS-CoV-2 induces the process of immunosenescence, increases endothelial cell damage, and leads to myofibrillar degradation and muscle wasting (sarcopenia) due to mitochondrial dysfunction and autophagy [102]

Conversely, IGF-1 facilitates FFA uptake into hepatocytes and adipocytes and stimulates lipogenesis, although its lipogenic effects are less pronounced [10, 11]

Whereas, in opposite to our theory, FAO suppression via Ranolazine has been demonstrated to attenuate AF occurrence in 1-week ACh-CaCl 2 -exposed rats ( Over-accumulation of lipid in cardiomyocyte cytosol occurs when FAs supply fails to match the needs created by FAs expenditure, thus is commonly observed in obesity (Figures 35)