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Deviations from this balance trigger a stress response, highlighting oxidative stress as a critical component of physiological redox signaling and control [5]

Upon reaction with HO , these radicals form 3-hydroxylysine, a neurotransmitter analogue 3,4 dihydroxyphenylalanine (DOPA) and, in interaction with another tyrosyl radical, forms a fluorescent protein crosslink dityrosine

Thus, MT and CK participate in a synergistic crosstalk mechanism under unfavorable environmental conditions

In particular, p53 inhibits SLC7A11 expression and reduces glutathione synthesis, making cancer cells susceptible to oxidative damage and sensitive to iron by increasing lipid peroxide levels [32,76,77]