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We found that the GGT deficient GGT enu1 mouse was protected against asthma in this model by an increased size of the extracellular LLF glutathione pool which buffered oxidant species derived from inflammatory cells, and preserved airway epithelial cell barrier function

Capitalizing on this, a ruthenium complex with an iodo, p-cymene, and phenylazopyridine ligand Ru(II)-OH was designed

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