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In the context of GBM, reducing iron levels may potentially decrease ferroptosis, rendering GBM cells more resistant to therapies that induce this form of cell death

By orchestrating the expression of target genes, it promotes cell cycle arrest, DNA repair, and apoptotic signaling transduction, thereby regulating cell division, preventing the propagation of DNA-mutated or damaged cells, and initiating apoptosis when required

This supports our findings where MTX injection evoked oxidative stress with augmented hepatic lipid peroxidation product while declining hepatic antioxidant capacity as depicted by lower GSH and SOD