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The results of the present study showed that the expression levels of C-type lectins were significantly down-regulated in both groups a and b after 96 h of residual chlorine stress compared to the control group, with no significant difference in expression levels in group c

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Mutations in SOD1 cause ALS in humans [113], and the overexpression of the ALS-linked mutant hSOD1 also causes an ALS-like phenotype in rodents [114]

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All participants voluntarily agreed to participate, understanding their right to withdraw at any stage without consequence

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