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The use of omics technologies and bioinformatics further accelerates the discovery and development of new drugs (Li and Zhang, 2013)

illustrated that PRL reduces the immune response to the effects of lipopolysaccharide (LPS)-induced inflammation by attenuating expression toll-like receptor 4 (TLR4) and nuclear factor kappa B cells (NF-B) with subsequent reduction release of pro-inflammatory cytokines, including TNF-, IL-1, and IL-6 [45]
Lohmann, P

Zakharkina, T
