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Description
However, its clinical translation is hindered by poor bioavailability and a paucity of large-scale randomized controlled trials
Intracellular ROS accumulation increases unstable iron and lipid peroxidation deposition through AMPK/NCOA4-mediated ferritinophagy, ultimately leading to ferroptosis and dysfunction in rat BMSCs
elegans and demonstrates its essential role in fasting-induced longevity
The reasons are biological, not cosmetic
