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Mechanistically, DHA and EPA attenuate post-stroke neuroinflammation by suppressing pro-inflammatory cytokine production, inhibiting microglial overactivation, and promoting the biosynthesis of specialized pro-resolving lipid mediators such as resolvins, protectins, and maresins (142)

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Vitamin E has been reported to decrease monocyte recruitment as a result of suppressed formation of foam cells and reduce expression of scavenger receptors (SRs) on macrophages (Zapolska-Downar et al