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Although high levels of ROS can induce DNA damage, lipid peroxidation, and protein dysfunction, low to moderate levels have been shown to act as key signaling molecules that promote cancer cell proliferation, stemness maintenance, and migratory capacity [35]

As such, it helps protect our cells from damage due to oxidative stress which can be caused by concussions and other injuries, mental stress, toxin exposure, infections, exercise, and aging

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Uterine-specific deletion of Arid1a compromises gland development, diminishes Foxa2 and Lif expression, and disrupts the LIF-STAT3-EGR1 pathway, leading to implantation failure and subfertility