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Researchers also explore its potential to modulate growth factors and repair mechanisms in response to injury
Taken together, these studies suggest that C-terminal truncated Syn variants may act as molecular decoy sequestering heterogeneous molecules, both proteins and membrane organelles, in their vicinity through nonfunctional interaction, leading to diffuse cellular aggregates or LB nucleation, as observed in various neurodegenerative disorders 83
Enzyme-driven oxygen-fuelled pathway selectivity of tyrosine-containing peptide oxidation evolution
An in vitro model of Fabry disease
