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Research in this area typically examines GHRH-receptor interaction models, downstream growth-hormone release pathways, and related molecular signaling in preclinical systems

It is thought that an important underlying pathogenetic basis for the metabolic syndrome is insulin resistance and accompanying compensatory hyperinsulinemia[301]

The SASP is the major mediator of the paracrine effects of senescent cells and drives secondary senescence

Consistent with these findings, GSH-depleted mice showed significantly decreased weights of liver and epididymal (visceral) white adipose tissue (eWAT) (Figure 2, D and E), while weights of inguinal (subcutaneous) white adipose tissue (iWAT), brown adipose tissue (BAT), or skeletal muscle remained unchanged (Supplemental Figure 2, FH)