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The pathogenesis of MS is attributed to mitochondrial damage and subsequent loss of energy in the CNS of susceptible individuals by oxidative stress, as evidenced by investigations [110,111,112]

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GrivaMLagoudakiRTouloumiONousiopoulouEKaralisFGeorgiouTet alLong-term effects of enriched environment following neonatal hypoxia-ischemia on behavior, Bdnf and synaptophysin levels in rat hippocampus: effect of combined treatment with G-Csf

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