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Indeed, animal model studies confirm that lipopolysaccharide- (LPS-) induced maternal immune activation leads to an increase of expression profiles of oxidative stress-related genes and a decrease of expression profiles of critical neurodevelopmental genes in the fetal brain [114]

Estrogen has an antioxidant function, and estrogen deficiency can inhibit the Nrf2/GPX4 pathway to induce ferroptosis (73)

Activation of this pathway leads to an increase in GPX4 protein expression, which in turn inhibits concomitant iron death and subsequent myocardial injury, consequently delaying the development of MI [118]

are critical in producing hyperalgesia