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This phenomenon is likely due to the anti-inflammatory effects of ferroptosis inhibitors, which target OC-STAMP through the inflammation-related RANKL pathway [30, 31], consequently decreasing the expression of OC-STAMP

Angiogenesis-independent endothelial protection of liver: role of VEGFR-1

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Renal cellular transport of exogenous glutathione: Heterogeneity at physiological and pharmacological concentrations
