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revealed that CKD patients had poorer clinical cardiovascular outcomes in association with enhanced inflammation and oxidative stress, as evidenced by their elevated MDA-modified LDL levels and peak serum C-reactive protein (CRP) [43]

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Therefore, we speculated that CsGSTs could bind to and activate the thiol group of GSH, and the S-glutathionylated xenobiotics would be sequentially processed to participate in the cold stress response

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