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The glucocorticoid receptor directly interferes with proinflammatory transcription factors, such as NF-B and AP-1, suppressing transcription of cytokines, chemokines, and their receptors (Liberman et al., 2007
Further, patients should be informed that, given the chronic nature of IC/BPS, the typical course involves symptom exacerbations and remissions
For instance, inhibition of miR-137 and miR-9 enhanced RSL3-induced ferroptosis in melanoma by targeting SLC5A1 and GOT1, respectively, in the glutamine metabolism pathway [20, 30]
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