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As oxidative stress increases, further stimulation of pro-inflammatory cytokines such as TNF- and IL-1 increases and contributes to synaptic dysfunction and neuronal degeneration ( These studies suggest that the loss of antioxidant defense is an early biochemical event rather than a late-stage consequence of neuronal death
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Adequate levels support normal energy metabolism, which may help reduce fatigue related to deficiency
10.1016/j.jnutbio.2013.05.007 70 JiaZ.YangF.LiuX.ZhangX.HuW.ShengZ
