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Similar content being viewed by others Introduction Proteotoxic stress induces conserved cell protection mechanisms such as the heat shock response (HSR) and the unfolded protein response of the endoplasmic reticulum (UPR ER ) to maintain proteostasis 1
Accordingly, the disadvantage of these models is the absence of a simple way to assess barrier function such as TEER measurement and complicated permeability screening (Cho et al., 2017)
