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Taken together, our results demonstrate that relative to the apoE3 isoform apoE4 exhibits less protection of neuronal cells against agonists that are of relevance in the pathogenesis of AD
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Upon exposure to n-3 PUFAs, Th17 cells generated reduced quantities of proinflammatory cytokines like IL-17, IL-22, and IL-23, whereas Treg cells demonstrated elevated production of anti-inflammatory molecules such as Foxp3 (44)
