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While semaglutide does not directly cause leg cramps through its pharmacological mechanism, several indirect pathways related to its effects and the physiological changes during treatment may potentially contribute to muscle cramping in susceptible individuals

The mechanisms underlying these potential synergistic effects remain under active investigation and may involve growth factor signaling, extracellular matrix synthesis, or anti-catabolic pathways in joint tissues

Characterizing rapid, activity-linked conformational transitions in proteins via sub-second hydrogen deuterium exchange mass spectrometry

Role of impaired glycolysis in perturbations of amino acid metabolism in diabetes mellitus