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Indeed, recent data suggest that a chronic elevation of homocysteine level might indirectly reduce methylation of DNA, RNA, proteins, and phospholipids through a concomitant increase in SAH level secondary to homocysteine-mediated reversal of the SAH hydrolase reaction (Yi et al, 2000)

These results also reinforce the importance of GSH in preventing APAP-induced hepatotoxicity

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LAMP2A monomers on this membrane are capable of receiving substrate proteins, and this interaction initiates the formation of the required polymeric complex for substrate translocation