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Consistently, a complementary strategy showed that in aged mice, where IGF-I levels are decreased, cardiac overexpression of IGF-I significantly improved cardiomyocyte contractile function[283], attenuating oxidative stress-mediated protein damage, normalizing Ca 2+ homeostasis, reversing age-related alterations in the expression of pro-apoptotic proteins, and decreasing the apoptosis rate[284]

fast export of GSH, breakdown by GGT, and fast reimport of cysteine

GSH concentration and xCT and Gpx4 expression were significantly decreased, and lipid peroxidation was enhanced in TGF-1-stimulated renal tubular cells

Cambridge, M.A: Ed