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Also, each monomer contained a conserved thioredoxin domain, which includes the GSH binding site

Additionally, the treated rats showed less mitochondrial structural damage, reduced ROS accumulation, increased ATP synthesis, and restored MMP levels, indicating that it effectively corrects mitochondrial morphological damage and dysfunction [74]

Importantly, the N-terminal catalytic GTPase-activating domain of ArfGAP1 (contained within residues 1-136), serves as a poor substrate of LRRK2 ( Fig

1-DNJs influence on cellular pathways extends to its role as an inhibitor of -glucosidase, a cell surface protein involved in glucose metabolism and insulin signaling (Figure 9)