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10.1158/1078-0432.CCR-19-0603 88 MeirowD.DorJ.KaufmanB.ShrimA.RabinoviciJ.SchiffE.et al (2007)
Consequently, these cytokines rely on signaling through the JAKSTAT pathway, making it a prominent focus in current studies of AA pathogenesis [113]
First, co-expression of wildtype USP7 (USP7-WT) with GATA1 increased the GATA1 level (Figure 3A)
However, their contribution is context-dependent, as genetic ablation of ABCC1 does not fully prevent apoptosis-associated GSH loss in certain models 43 , indicating the existence of compensatory or alternative efflux mechanisms
