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Because folate status is a major determinant of blood homocysteine concentrations, the possible contribution of elevated homocysteine to the pathogenesis of NTDs has been postulated

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151 Although the focus of this review is on oral supplementation with SA, it is nevertheless worth mentioning that the administration of high doses of intravenous (IV) vitamin C in those with glucose-6-phosphate dehydrogenase (G6PD) deficiency is controversial, 152 as it may pose the risk of triggering hemolysis

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