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The overexpression of SNHG14 inhibits the expression of miR-483-5p to promote the HDAC4 expression, ROS production, and the inflammatory factor and ECM protein secretion, ultimately exacerbating HG-induced DKD (Huang et al., 2024)
This can lead to steadily increasing activation of the SAM system

Common non-clinical applications include analytical reference work, stability studies, and controlled in-vitro research workflows
