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These discrepancies point to the fact that the activity of gut metabolites and signaling mediators is highly contextdependent, influenced by variables such as dietary inputs, gut microbiota composition, host health status, and metabolic individuality

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Although no in vivo experiments have been reported, a mutant strain incapable of producing CD0873 is unable to bind Caco2 cells, suggesting a role in adhesion to enteric cells

TcdA induces inflammation by releasing neuropeptides like Substance P (SP) and Calcitonin gene-related peptide (CGRP) from sensory neurons, enhancing the inflammatory signal [174,175] (Figure 3)