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Description
Extrinsic apoptotic pathways are activated by interactions between cell surface exposed death-inducing ligands such as FAS ligands (FASL) and tumor necrosis factor (TNF), and their cognate receptors Fas cell surface death receptor (FAS) and TNF receptor (TNFR) [110, 111]
It has been shown to be effective in murine models of rheumatoid arthritis and may be beneficial in other chronic inflammatory diseases
Sun J, Zhou C, Zhao Y, Zhang X, Chen W, Zhou Q, et al
Raffa, R
