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This pathological state hinders epithelialization and vascularization, enabling biofilm-forming pathogens such as Staphylococcus aureus and Pseudomonas aeruginosa to dominate the wound microenvironment [7]
Ghitti, M
In CRC cells, the mutant isocitrate dehydrogenase 1 inhibitor Ag120 inhibited ASCT2-mediated Gln uptake, leading to decreased cell proliferation and increased oxidative stress both in vitro and in vivo [63]
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