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Although the mitochondria-independent pathway driven by GPX4 inhibitors is crucial for understanding and improving cancer treatment, -dependent ferroptosis induced by cysteine deficiency or GSH depletion may be more relevant to the pathophysiological context of chronic human diseases
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doi:10.1080/13625187.2016.1263292
The opposite face of this pentraxin is the effector face, where complement C1q binding occurs and also Fc receptors are supposed to bind
