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Importantly, MKR mice exhibit defects in the free fatty acid (FFA) oxidation pathway, which leads to elevations in serum FFAs and triglycerides (TGs), as well as increased TG deposits in liver and muscle tissues, suggesting that hyperlipidemia and accumulated lipids in tissues may be causative factors for the progression of type 2 diabetes in MKR mice, replicating human cases with inherited lipodystrophy or dyslipidemia (37, 38)

As direct consequence, some SnCE1 acetylation sites are enzymatically deacetylated upon exposure in the host cell following injection of SnCE1 while others might remain stable as they are not accessible for enzymatic deacetylation

96 For instance, Liu et al

Consensus recommendations 5.1.1 Simplified score 2008 score should be used for the diagnosis of pediatric autoimmune hepatitis (LOE 3, Strong recommendation)