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These antibody-based therapies, however, differ in their ability to induce direct apoptosis, mount complement-mediated cytotoxicity (CDC), inhibit CD38 directly, and induce antibody-dependent cell-mediated phagocytosis (ADCP) (Table 1) (76, 96)

The reasoning is simple: when oral bacteria run unchecked, they can trigger inflammatory responses in the body

Analysis of conjunctival sac microbiome in dry eye patients with and without Sjgren's syndrome

Another approach of TNF- pathway blockade that has been recently examined is the development of orally active small molecules (such as UCB-9260) that destabilize TNF- trimer, impeding TNF--TNFR1 binding and signaling [200]