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Thus, the causal relationship among ER stress induction, pERK1/2, and autophagy activation was examined using MAPK/ERK and ER stress inhibitors, namely U0126 and 4-PBA

Also, several nuclear-encoded components of a putative mitochondrial pathway for de novo fatty acid synthesis were identified and characterized within mitochondria

In contrast, in obese human subjects under fasting conditions, the increase in urinary excretion of acetylcarnitine was markedly slower and at a lower level, suggesting a slower formation of acetyl-CoA derived from fat oxidation

T., Calogero, A
